FSGS represents one of the most common hallmarks of podocyte stress and damage. To this date, the underlying mechanisms specifically in primary and recurrent FSGS are poorly understood. Via the analysis of patient samples obtained from very well defined cohorts in combination with novel in vitro and in vivo disease modeling, we aim to uncover conserved molecular pathways altered during the course of Focal Segmental Glomerulosclerosis.


Researchers:

Fabian Braun

Inka Homeyer






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